Data: 08/08/2026
Total encontrado: 4
1/4US=85 / Biol/Mol
Hwang JT, Lee YK, Shin JI, Park OJ. Anti-inflammatory and anticarcinogenic effect of genistein alone or in combination with caspsaicin in TPA-treated rat mammary glans or mammary cancer cell line. In: Diederich M. Natural compounds and their role in apoptotic cell signaling pathways. Boston: Blackwell, 2009. p. 415-20. (Annals of the New York Academy of Sciences Vol 1171) ISBN 978-1-57331-737-5.

TRATAMENTO DE COMBINACAO DE GENISTEINA E DE CAPSAICINA; APOPTOSE; COX-2; AMPK; EFEITOS ANTI-INFLAMATORIOS E ANTICARCINOGENICA


2/4US=85 / Biol/Mol
Lee YK, Park SI, KimYM, Park OJ. Regulatory effect of the AMPK-COX-2 signaling pathway in curcumin-induced apoptosis in HT-29 colon cancer cells. In: Diederich M. Natural compounds and their role in apoptotic cell signaling pathways. Boston: Blackwell, 2009. p. 489-94. (Annals of the New York Academy of Sciences Vol 1171) ISBN 978-1-57331-737-5.

APOPTOSE; CURCUMINA; AKT; AMPK; COX-2; PARADA DO CICLO CELULAR; HT-29COLON CELULAS CANCEROSAS; P53 TIPO SELVAGEM


3/4US=85 / Biol/Mol
Park IJ, Lee YK, Hwang JT, Kwon DY, Ha J, Park OJ. Green tea catechin controls apoptosis in colon cancer cells by attenuation of H2O2-stimulated COX-2 expression via the AMPK signaling pathway at low-dose H2O2. In: Diederich M. Natural compounds and their role in apoptotic cell signaling pathways. Boston: Blackwell, 2009. p. 538-44. (Annals of the New York Academy of Sciences Vol 1171) ISBN 978-1-57331-737-5.

EGCG; COX-2; AMPK; HT-29 CANCER DE CELULAS DO COLON; BAIXAS DOSES DE H2O2; APOPTOSE; P53 TIPO SELVAGEM


4/4US=22-C / Câncer
Horn L, Araújo LHL, Nana-Sinkam P, Otterson GA, Williams TM, Carbone DP. Biologia molecular do câncer de pulmão. In: DeVita VT, Lawrence TS, Rosenberg SA. DeVita, Hellman, and Rosenberg’s cancer: principles & practice of oncology. 10 ed. Philadelphia: Wolters Kluwer, 2015. p. 482-494. ISBN 978-1-45119294-0.

SUSCEPTIBILIDADE AO CANCER DE PULMAO; SUSCETIBILIDADE GENETICA E SUBSTANCIAS CANCERIGENAS NA FUMACA DO TABACO; ALTERACOES MOLECULARES EM PRENEOPLASIA; ALTERACAO GENETICA E EPIGENETICA NO CANCER DE PULMAO; PROTOONCOGENESE; FATOR DE TERAPIAS DIRECIONADAS; TECNOLOGIA GENOMICA NA ANALISE DO CANCER DE PULMAO; GENES SUPRESSORES DE TUMORES. SUPRESSAO DO CRESCIMENTO; CICLINAS E VIAS REGULADORAS DO CICLO CELULAR; LKB1; AMPK; VIAS MTOR; OUTROS SUPRESSORES TUMORAIS PUTATIVOS; OUTRAS ANORMALIDADES BIOLOGICAS NO CANCER DE PULMAO; MICRORNAS EM CANCER DE PULMAO. DIAGNOSTICO; MICRORNAS EM CANCER DE PULMAO. TERAPIA; INVASAO, METASTASE E ANGIOGENESE; CANCER DE CELULAS TRONCO. HIPOTESIS; FERRAMENTAS MOLECULARES NA CLINICA DO CANCER DO PULMAO