Data: 08/08/2026
Total encontrado: 41
1/41US=200 / Tese/FAP
Campos ECR. Análise da expressão do gene PTEN e da proteína akt associada a fatores clínicos e anatomopatológicos como prognósticos no carcinoma de células renais. São Paulo, 2007. 101 p. Dissertação(Mestrado)-Fundação Antônio Prudente.

Disponível em: http://accamargo.phlnet.com.br/MESTRADO/2007/EuricoCletoRibeirodeCampos/EuricoCletoRibeiroCamposmestrado.pdf

Curriculum Lattes

CANCER RENAL; EXPRESSAO GENICA; PROGNOSTICO; IMUNOHISTOQUIMICA; ESTADIAMENTO DO CANCER


2/41US=316 / Tese/FAP
Caly DN. Melanoma acral-lentiginoso: análise de proteínas e genes do ciclo celular nas vias de sinalização p16/prb, p53 e PTEN. [Acral lentinous melanoma: protein and cell cycle gene analysis of p16/pRb, p53 and PTEN pathways]. São Paulo, 2010. 63 p. Tese(Doutorado)-Fundação Antônio Prudente.

Disponível em: http://accamargo.phlnet.com.br/Doutorado/2010/DCali/DCali.pdf

Curriculum Lattes

MELANOMA MALIGNO; IMUNO-HISTOQUIMICA; GENES P16; GENES P53; PROTEINA DE PROTO-ONCOGENE AKT; CANCER DA FARINGE; CANCER DA LARINGE; COMPLICACOES POS-OPERATORIOS


3/41US=333 / Tese/FAP
Castro CV. Estudo de receptores de tirosina quinase em cordomas. [Study of receptors tyrosine kinase in chordomas]. São Paulo, 2011. 85 p. Dissertação(Mestrado)-Fundação Antônio Prudente.

Disponível em: http://accamargo.phlnet.com.br/MESTRADO/2011/CarolCastro/CarolCastro.pdf

Curriculum Lattes

CORDOMA; PROTEINAS TIROSINA QUINASES; CANCER; NEOPLASIAS; MARCADORES BIOLOGICOS; BIOLOGIA MOLECULAR


4/41US=335 / Tese/FAP
Machado CF. Estudo funcional de mutantes da proteína prion celular associados às doenças por prion. [Functional study of mutant cellular prion protein associated with prion diseases]. São Paulo, 2011. 139 p. Tese(Doutorado)-Fundação Antônio Prudente.

Disponível em: http://accamargo.phlnet.com.br/Doutorado/2011/CMachado/CMachado.pdf

Curriculum Lattes

PRIONS. PATOGENECIDADE; DOENCAS DE PRION; LAMININA; MUTACAO; POLIMORFISMO GENETICO


5/41US=348 / Tese/FAP
Chiappini PBO. Estudo da expressão de mTOR, PTEN e AKT na mucosa gástrica normal e carcinomas gástricos. [Study of the expression of mTOR, PTEN and AKT normal mucosa and gastric carcinomas]. São Paulo, 2011. 86 p. Dissertação(Mestrado)-Fundação Antônio Prudente.

Disponível em: http://accamargo.phlnet.com.br/MESTRADO/2011/PaulaChiappini/PaulaChiappini.pdf

Curriculum Lattes

NEOPLASIAS GASTRICAS; MARCADORES BIOLOGICOS; IMUNOISTOQUIMICA; PTEN FOSFOIDROLASE; PROTEINAS PROTO-ONCOGENICAS C-AKT; MTOR


6/41US=94 / Biol/Mol
Matthews DJ, Gerritsen ME. Intracellular signal transduction cascades. In: ___. Targeting protein kinases for cancer therapy. New Jersey: Wiley, 2010. p. 265-325. ISBN 978-0-470-22965-1.

TRANSDUCAO DE SINAIS INTRACELULARES; PI3K; FOSFATIDILINOSITOL 3-QUINASE; PTEN; PDK1; DESIDROGENASE PIRUVATO LIPOAMIDE QUINASE ISOENZIMA 1; AKT; CANCER; CANCER DE MAMA; CANCER COLORRETAL; CANCER ENDOMETRIAL; CARCINOMA GASTRICO; GLIOBLASTOMA; CARCINOMA HEPATOCELULAR; CANCER DE PULMAO; MELANOMA; CANCER DE OVARIO; CANCER DE PANCREAS; CARCINOMA DE PROSTATA; CARCINOMA DE TIREOIDE; NEOPLASIAS HEMATOLOGICAS; MTOR; QUINASE P70S6; PROTEINA QUINASE ATIVADA POR MITOGENO (MAPK); ERK; FAMILIA QUINASE RAF; MEK; PIM; PROTEINA QUINASE C (PKC)


7/41US=32 / Gastro
Huitzil-Melendez FD, Abou-Alfa GK, Morse MA. Novel therapies targeted at sinal transduction in liver tumors. In: Clavien PA, Breitenstein S. Malignant liver tumors: current and emerging therapies. 3ª ed. New Jersey: John Wiley & Sons, 2010. p. 382-92. ISBN 978-1-4051-7976-8.

CANCER DE FIGADO; TRANSDUCAO DE SINAL; RECEPTORES TIROSINA QUINASE (RTK); RECEPTOR DO FATOR DE CRESCIMENTO EPIDERMICO; FATOR DE CRESCIMENTO INSULIN-LIKE (IGF-I); PROTO-ONCOGENE C-MET; VIAS INTRACELULARES DE TRANSDUCAO DE SINAL; MAPK; PI3K; AKT; MTOR; FAMILIA RAS; WNT/B-CATENIN; ANGIOGENESE


8/41US=2 / Farmacologia
Romanelli RJ, Williams JT, Neve KA. Dopamine receptor signaling: intracellular pathways to behavior. In: Neve KA. The dopamine receptors. 2ªed. New York: Humana Press, 2009. p. 137-73. ISBN 978-1-60327-332-9.

SINALIZACAO DO RECEPTOR DE DOPAMINA; VIAS INTRACELULARES PARA O COMPORTAMENTO; ADENILATO CICLASE; PROTEINA G; ARRESTINA; AKT; FOSFOLIPASE C; KINASE PROTEINA A; AMP CICLICO


9/41US=6 / Quimioterapia
Airley R. Inhibitors of the AKT/PKB pathway. In: ___. Cancer chemotherapy: basic science to the clinic. New Jersey: John Wiley & Sons, 2009. p. 229-31. ISBN 978-0-470-09254-5.

INIBICAO DA VIA AKT/PKB


10/41US=22 / Urologia
Cho D, Mier JW, Atkins MB. PI3K/AKt/mTOR pathway: a growth and proliferation pathway. In: Bukowski RM, Figlin RA, Motzer RJ. Renal cell carcinoma: molecular targets and clinical applications. 2ªed. New York: Humana Press, 2009. p. 267-85. ISBN 978-1-58829-737-2.

INIBIDORES PI3K; RCC; HIPERATIVACAO DO AKT/PI3K; ATIVIDADE AKT; TEMSIROLIMUS


11/41US=85 / Biol/Mol
Lee YK, Park SI, KimYM, Park OJ. Regulatory effect of the AMPK-COX-2 signaling pathway in curcumin-induced apoptosis in HT-29 colon cancer cells. In: Diederich M. Natural compounds and their role in apoptotic cell signaling pathways. Boston: Blackwell, 2009. p. 489-94. (Annals of the New York Academy of Sciences Vol 1171) ISBN 978-1-57331-737-5.

APOPTOSE; CURCUMINA; AKT; AMPK; COX-2; PARADA DO CICLO CELULAR; HT-29COLON CELULAS CANCEROSAS; P53 TIPO SELVAGEM


12/41US=85 / Biol/Mol
Kim SH, Kim SH, Kim YB, Jeon YT, Lee SC, Song YS. Genistein inhibits cell growth by modulating various mitogen-activated protein kinases and AKT in cervical cancer cells. In: Diederich M. Natural compounds and their role in apoptotic cell signaling pathways. Boston: Blackwell, 2009. p. 495-500. (Annals of the New York Academy of Sciences Vol 1171) ISBN 978-1-57331-737-5.

GENISTEINA; ERK1/2; P38; MARK; JNK; AKT


13/41US=49-F / Câncer
Jiang BH,Liu LZ. PI3K/PTEN signaling in angiogenesis and tumorigenesis. In: Woude GFV, Klein G. Advances in cancer research. Amsterdam: Elsevier, 2009. p. 19-65. ISBN 978-0-12-3744371.

PI3K/PTEN; SINALIZACAO NA ANGIOGENESE; SINALIZACAO TUMORIGENESE; ANGIOGENESE REGULADA POR VEGF; ABERRACOES GENETICAS DE PI3K, PTEN E AKT EM CANCER; PAPEIS DE PI3K E AKT NA REGULACAO DA ANGIOGENESE; CRESCIMENTO DO TUMOR; METASTASE DE TUMOR; ANGIOGENESE TUMORAL


14/41US=14 / Câncer
Thayyullathil F, Chathoth S, Hago A, Wernery U, Patel M, Galadari S. Investigation of heat stress response in the camel fibroblast cell line dubca. In: Branicki FJ, Adem A, Ghazal-Aswad S, Torab FC. Recent advances in clinical oncology. Boston: Blackwell, 2008. p. 376-84. ISBN 978-1-57331-700-9.

ESTRESSE POR CALOR; AKT; FIBROBLASTOS CAMELO; LINHA CELULAR DUBCA; PROTEINA DE CHOQUE TERMICO; JNK; C-JUN


15/41US=14 / Câncer
Galadari S, Thayyullathil F, Hago A, Wernery U, Patel M, Chathoth S. Akt depletion is an important determinant of L929 cell death following heat stress. In: Branicki FJ, Adem A, Ghazal-Aswad S, Torab FC. Recent advances in clinical oncology. Boston: Blackwell, 2008. p. 385-92. ISBN 978-1-57331-700-9.

RESPOSTA DO ESTRESSE POR CALOR; ESGOTAMENTO AKT; L929; MORTE CELULAR


16/41US=14 / Câncer
Choi JH, Yang YR, Lee SK, Kim SH, Cha JY, Oh SW, Ha JR, Ryu SH, Suh PG. Potential inhibition of PDK1/Akt signaling by phenothiazines supresses cancer cell proliferation and survival. In: Branicki FJ, Adem A, Ghazal-Aswad S, Torab FC. Recent advances in clinical oncology. Boston: Blackwell, 2008. p. 393-403. ISBN 978-1-57331-700-9.

FENOTIAZINAS; PDK1; AKT; CELULAS CANCEROSAS; APOPTOSE; PROLIFERACAO


17/41US=35 / Biol/Mol
Dorsey FC, Steeves MA, Cleveland JL. Apoptosis, autophagy, and necrosis. In: Mendelsohn J, Howley PM, Israel MA,Gray JW, Thompson CB. The molecular basis of cancer. 3rd ed. Philadelphia: Saunders Elsevier, 2008. p. 205-20. ISBN 978-1-4160-3703-3.

APOPTOSE; AUTOFAGIA; NECROSE; CASPASES; FAMILIA BCL-2; MORTE CELULAR; NF-KEB; FATOR NUCLEAR KAPPA B; AKT/PTEN; ESTRESSE ONCOGENICO; P53


18/41US=39 / Câncer
McCubrey JA, Franklin RA, Bertrand FE, Taylor JR, Chappell WH, Sokolosky ML, Wong EWT, Abrams SL, Stadelman KM, Misaghian N, Ludwing DL, Basecke J, Libra M, Stivala F, Milella M, Tafuri A, Martelli AM, Lunghi P, Bonati A, Terrian DM, Lehmann BD, Steelman LS. Targeting survival cascades induced by activation of Ras/Raf/MEK/ERK and PI3K/Akt pathways to sensitize cancer cells to therapy. In: Bonavida B. Sensitization of cancer cells for chemo/immuno/radio-therapy. Los Angeles: Humana Press, 2008. p. 81-114. ISBN 978-1-58829-989-5.

SEGMENTACAO CASCATAS DE SOBREVIVENCIA; ATIVACAO DE RAS / RAF / MEK / ERK; VIAS PI3K/AKT; SENSIBILIZACAO DAS CELULAS CANCEROSAS; APOPTOSE; CANCER HEMATOPOIETICO


19/41US=64 / Câncer
Harvey RD, Silberman J, Lonmial S. The PI3 kinase/akt pathway as a therapeutic target in multiple myeloma. In: Lonial S. Mylenoma therapy: pursuing the plasma cell. 5th ed. New Jersey: Humana Press, 2008. p. 309-22. (Comtemporary Hematology) ISBN 978-1-934115-82-4.

PI3 QUINASE; AKT; TERAPEUTICO NO MIELOMA MULTIPLO; MIELOMA MULTIPLO. TERAPIAS


20/41US=58 / Câncer
Lee MH, Yeung SCJ, Yang HY. Interplay of 14-3-3 family of proteins with DNA damage-regulated molecules in checkpoint control. In: Siddik ZH. Checkpoint controls and targets in cancer therapy. New Jersey: Humana Press, 2008. p. 69-80. (Cancer Drug Discovery and Development Series) ISBN 978-1-60761-177-6.

FAMILIA DE PROTEINAS COM 14-3-3; DANOS AO DNA; RESPOSTA CHECKPOINT; 14-3-3; AKT; MDM2; P53; DANOS NO DNA


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